Saving Babies from the Threat of Microplastic Toxins we Need to Stop Consume Plastik
Nina : We want a strong treaty to protect human health and the environment by reducing plastic production, eliminating threats from toxic chemicals throughout the plastic life cycle
“The Mother's Womb Has Been Contaminated With Microplastics, Now The Safest Place For Humans Has Been Contaminated. Where else is a safe place for humans?”Aeshnina Said.
Recent research has found the presence of microplastics in the bodies of babies, from the placenta to breast milk. The microplastics found in humans come from the consumption of food and beverages packaged in single-use plastic, air contaminated with microplastics, and skin contact with personal care products containing microplastics (microbeads). This situation is deeply concerning, as the 16,000 types of chemicals in plastics, including toxic substances like Bisphenol A (BPA), phthalates, PCBs, and PBDEs, have been proven to negatively impact human health. Exposure to these chemicals during pregnancy and early life can interfere with growth, neural development, and the reproductive system.
Microplastics
can be absorbed through the digestive tract via paracellular absorption and
phagocytosis, entering the bloodstream. Subsequently, microplastic particles
smaller than 20 micrometers are distributed to secondary organs, such as
muscles, liver, kidneys, heart, brain, breast milk, and even the placenta of
the fetus.
Microplastics
that reach the placenta can potentially enter the amniotic fluid and be
absorbed by the fetus.
1.Placenta—
A study by Braun et al. (2021) identified various types of microplastics in the
placentas of pregnant women, including PP (33%), PVC (26.67%), PET (16.67%),
and HDPE (10%). Further research showed that microplastic exposure in the
placenta increased over the past 15 years, from 60% in 2006 to 100% in 2021
(Weingrill et al., 2023). This is alarming, as the placenta plays a crucial
role in connecting the mother and fetus. Microplastic exposure could disrupt
hormone balance and threaten fetal organ development[1].
2.
Amniotic
Fluid — Amniotic fluid, which protects the fetus during pregnancy, also
contains microplastics. Halfar et al. (2023) found that 72.72% of microplastics
in the amniotic fluid were PET, with potential biological impacts such as
preterm birth and stunted fetal growth.[2]
3.
Meconium
(First Stool of the Baby) —
Microplastics detected in meconium, including other types (82.8%), PET (4.17%),
and PVC (2.2%) (Li et al., 2023), suggest exposure during fetal development.
This exposure likely comes from the transfer of microplastics from the mother
to the fetus via the placenta. Since meconium starts to form at around 16 weeks
of pregnancy, the presence of microplastics in meconium indicates exposure
during fetal development.[3]
4.
Infant
Feces — In a study by Li et al. (2023), infant feces contained 76.32%
of "other" types of microplastics and 7.86% PET. The presence of
microplastics in infant feces is linked to environmental exposure after birth,
such as through breast milk, formula milk, plastic feeding bottles, or plastic
toys. This study found that babies who frequently used plastic bottles and
sucked on plastic toys had higher microplastic levels in their feces.[4]
5.
Breast
Milk — A study by Ragusa et al. (2022) reported microplastic
contamination in breast milk, including PET (37.50%) and PP (16.67%). The
chemicals in microplastics can enter breast milk, carrying risks of endocrine
disruption, which could impact the baby's neurocognitive development.
Lipophilic microplastics can bind to fat components in breast milk, increasing
the risk of accumulation in the baby's body. This means that microplastics may
not only be present as free particles in breast milk but also embedded in fat
components, which are then consumed by the baby.[5]
Microplastics have the ability to absorb and bind harmful pollutants from their surroundings. The microplastics and the chemicals they contain are bioaccumulative and persistent, meaning they accumulate in the tissues of organisms over their lifetime because the body cannot fully eliminate them. Additionally, they have biomagnification potential, meaning microplastics and their chemicals move from one organism to another in the food chain, leading to higher concentrations in top predators, including humans. Exposure to microplastics and their thousands of chemical compounds presents serious risks to developing babies. These include inflammation, immune system disruption, and interference with brain and nerve development. Microplastics may also increase the risk of infections and affect long-term health.This research calls for urgent attention and decisive action to limit the use of single-use plastics and improve the safety standards for plastic packaging materials used daily. A concerted effort from the government, industry, and society at large is crucial to protect babies and future generations from this growing threat.
[1] Braun, T., Ehrlich, L., Henrich, W., Koeppel, S., Lomako, I.,
Schwabl, P., & Liebmann, B. (2021). Detection of microplastic in human
placenta and meconium in a clinical setting. Pharmaceutics, 13(7),
921.
[2] Halfar, J., Čabanová, K., Vávra, K., Delongová, P., Motyka, O., Špaček, R., ... & Heviánková, S. (2023). Microplastics and additives in patients with preterm birth: The first evidence of their presence in both human amniotic fluid and placenta. Chemosphere, 343, 140301.
[3] Liu, S., Guo, J., Liu, X., Yang, R., Wang, H., Sun, Y., ... & Dong, R. (2023). Detection of various microplastics in placentas, meconium, infant feces, breastmilk and infant formula: A pilot prospective study. Science of The Total Environment, 854, 158699.
[4] Liu, S., Guo, J., Liu, X., Yang, R., Wang, H., Sun, Y., ... & Dong, R. (2023). Detection of various microplastics in placentas, meconium, infant feces, breastmilk and infant formula: A pilot prospective study. Science of The Total Environment, 854, 158699.
[5] Ragusa, A., Svelato, A., Santacroce, C., et al. (2021). Plasticenta: First evidence of microplastics in human placenta. Environment International, 146, 106274. DOI: 10.1016/j.envint.2020.106274.
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